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SOCS2andSOCS3areRelatedtoMyoblastDifferentiation
viatheIGF1andGHSignalingTransductionSystem
LIUWucyi,ZHAOChunjiang,WUChangxin
LCoforrafnpmalimeeangTeunohgyChnaprestoralUneeonBujireIoooii
2FemngToucionCuieofeeneodiiPoeaeag0tD
AbstractAneendbdocniednilyolprosinneuuppeneraleyelieaigelngGOCSnipvtis
gedbekcopnmgiegowcoriecingorcyolneneJnonhnacealimerduee
erofnenpinOAKSTVTnnioaghhnudporedndnedeeota
20CSiphnieynicinminbiotdtfoeniionnduppresinetinedeilirnnetouddlarintunmaeom
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20CSieeCFJGolniksromtiteorsndGHpowblomeeeelngurenepeidlyene
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dS0csimyepoeilinesethrpan
bnooseienngtteconminclmdcdevipmeae
nnsetonthegowtsteersiuineand0cSaptaty
KeyworesocssocyaS0cs4rGFJ1IGFiGHJAKSTATPadwaynyogoes
CLCnambervansDecumentcod
ARamilyf30CSeuppressorofeyiokinesig
nngproksinshavingasinilardomainstrueture
basreeendyeenmuchdeseribedTheSOCSami
jycurrentyindudesaghtmembe,CIScr
tokineindusitleSH2contsiningproiinand
S0CStnS0CS10Theircommondomsin
ctoreconsietsotanierminalgonofvar
ableengthandaminoidcomposiionacentnal
SH2domainandaCherminaldomincontsininge
conservedS0CSbosAsidsfromthefournen
barsotheaarlydentiedaoppressorotcrokines
sgralagfamiySOCS1,,50CS3,and
CIS,Rionstal88haveearhedDNA
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